Associate Professor
Anita Ryningen
Publications
-
Acute response in circulating microRNAs following a single bout of short-sprint and heavy strength training in well-trained cyclists
-
p53 Protein Isoform Profiles in AML: Correlation with Distinct Differentiation Stages and Response to Epigenetic Differentiation Therapy
-
A pilot study of circulating monocyte subsets in patients treated with stem cell transplantation for high-risk hematological malignancies
-
Immunomodulatory drugs alter the metabolism and the extracellular release of soluble mediators by normal monocytes
-
Open online E-learning resources at epraksis.no as preparation for hands-on laboratory practice
-
Circulating monocyte subsets in multiple myeloma patients receiving autologous stem cell transplantation – a study of the preconditioning status and the course until posttransplant reconstitution for a consecutive group of patients
-
Standardization of sampling and sample preparation for analysis of human monocyte subsets in peripheral blood
-
Power production and biochemical markers of metabolic stress and muscle damage following a single bout of short-sprint and heavy strength exercise in well-trained cyclists
-
The effect of pre-analytical factors on circulating monocyte subpopulations
-
Connexin expression in human acute myeloid leukemia cells: Identification of patient subsets based on protein and global gene expression profiles.
-
Inhibition of Mammalian target of rapamycin in human acute myeloid leukemia cells has diverse effects that depend on the environmental in vitro stress
-
Connexin expression by human primary AML cells
-
Connexin expression by human primary AML cells
-
Depletion of the human N-alpha-terminal acetyltransferase A induces p53-dependent apoptosis and p53-independent growth inhibition
-
Hypoxia increases HIF-1 alpha expression and constitutive cytokine release by primary human acute myeloid leukaemia cells
-
PHARMACOLOGICAL TARGETING OF THE PI3K-AKT/PKB-MTOR PATHWAY ALTERS LOCAL ANGIOREGULATION IN ACUTE MYELOGENOUS LEUKEMIA
-
The chaperone-like protein HYPK acts together with NatA in cotranslational N-terminal acetylation and prevention of Huntingtin aggregation
-
The Chaperone-like protein HYPK acts together with NatA in cotranslational N-terminal acetylation and prevention of Huntingtin aggregation
-
The human protein N-a-acetyltransferase complex hNatA and HYPK. Affecting Htt-Aggregation
-
The chaperone-like protein HYPK and NatA prevents Huntingting aggregation
-
The chaperone-like protein HYPK and NatA prevents Huntingtin aggregation
-
The chaperone-like protein HYPK acts together with NatA in cotranslational N-terminal acetylation and prevention of Huntingtin aggregation
-
Knockdown of the human N-alpha-acetyltransferace complex C (hNatC) leads to p53-dependent apoptosis and aberrant hArl8b localization
-
A subset of patients with high-risk acute myelogenous leukemia shows improved peripheral blood cell counts when treated with the combination of valproic acid, theophylline and all-trans retinoic acid
-
Long-term cryopreservation of autologous stem cell grafts: a clinical and experimental study of hematopoietic and immunocompetent cells
-
Use of different DMSO concentrations for cryopreservation of autologous peripheral blood stem cell grafts does not have any major impact on levels of leukocyte- and platelet-derived soluble mediators
-
The protein kinase C agonist PEP005 increases NF-kB expression, induces differentiation and increases constitutive chemokine release by primary acute myeloid leukaemia cells
-
The human N-acetyltransferase complex C (hNatC)
-
N-terminal protein acetylation in humans
-
Identification and characterization of the human N-acetyltransferase complexes hNatB and hNatC
-
Circulating T cells derived from acute leukemia patients with severe therapy-induced cytopenia express a wide range of chemokine receptors
-
Chemokine receptor expression by circulating T cells derived from acute leukemia patients with severe chemotherapy-induced cytopenia express a wide range of chemokine receptors
-
Release of angiopoietin-1 by primary human acute myelogenous leukemia cells is associated with mutations of nucleophosmin, increased by bone marrow stromal cells and possibly antagonized by high systemic angiopoietin-2 levels
-
In vitro induction of a dendritic cell phenotype in primary human acute myelogenous leukemia (AML) blasts alters the chemokine release profile and increases the levels of T cell chemotactic CCL17 and CCL22
-
In vivo biological effects of ATRA in the treatment of AML
-
Identification of the human N-alpha-acetyltransferase complex B (hnatB): a complex important for cell-cycle progression
-
The novel hNatB complex is important for cell cycle progression
-
Functional characteristics and gene expression profiles of primary acute myeloid leukaemia cells identify patient subgroups that differ in the susceptibility to histone deacetylase inhibitors
-
Linking susceptibility to histone deactylase inhibitors and basal gene expression profiles in acute myeloid leukaemia
-
The protein kinase C activator PEP005 increases the chemokine release in native acute myelogenous leukemia (AML) blasts
-
The proteasome inhibitors bortezomib and PR-171 have antiproliferative and proapoptotic effects on primary human acute myeloid leukaemia cells
-
Subclassification of patients with acute myelogenous leukemia based on chemokine responsiveness and constitutive chemokine release by their leukemic cells
-
Clonogenic acute myelogenous leukemia cells are heterogeneous with regard to regulation of differentiation and effect of epigenetic pharmacological targeting
-
Functional characteristics and gene expression profiles of primary acute myeloid leukamia cells indentify patient subgroups that differ susceptibility to histone deactylase inhibitors
-
Histone deacetylase inhibitors in cancer treatment: a review of the clinical toxicity and the modulation of gene expression in cancer cells
-
Epigenetic targeting in acute myeloid leukemia: use of flow cytometry in monitoring therapeutic effects
-
Development and evaluation of labeled antibodies as optical contrast reagents in acute myeloid leukemia (AML)
-
Protein lysine acetylation in normal and leukaemic haematopoiesis: HDACs as possible therapeutic targets in adult AML
-
Treatment with valproic acid, all-trans retinoic acid (ATRA) and theophyllamine for 9 days caused a persistent increase in peripheral blood platelet counts for a patient with acute myelogenous leukemia
-
Acute myelogenous leukemia in a patient with Li-Fraumeni syndrome treated with valproic acid, theophyllamine and all-trans retinoic acid: a case report
-
T lymphocyte chemotactic chemokines in acute myelogenous leukemia (AML): local release by native human AML blasts and systemic levels of CXCL10 (IP-10), CCL5 (RANTES) and CCL17 (TARC)
-
Induction of apoptosis in human cells by RNAi-mediated knockdown of hARD1 and NATH, components of the protein N-alpha-acetyltransferase complex
-
Microvascular endothelial cells increase proliferation and inhibit apoptosis of native human acute myelogenous leukemia blasts
-
Reticulated platelets are increased in chronic myeloproliferative disorders, pure erythrocytosis, reactive thrombocytosis and prior to hematopoietic reconstitution after intensive chemotherapy
-
Stress-induced in vitro apoptosis of native human acute myelogenous leukemia (AML) cells shows a wide variation between patients and is associated with low BCL-2 : Bax ratio and low levels of heat shock protein 70 and 90
-
Biological function of N-alpha-acetyltransferase A complex in human cells
-
Downregulation of hARD1-NATH, components of N-alpha-acetyltransferase complex, triggers apoptosis
-
In vitro crosstalk between fibroblasts and native human acute myelogenous leukemia (AML) blasts via local cytokine networks results in increased proliferation and decreased apoptosis of AML cells as well as increased levels of proangiogenic Interleukin 8